The study in question is Dietary Carbohydrate restriction as the first approach in diabetes management. Critical review and evidence base. Here are my comments on the 12 points.
Point 1 is wrong. Hyper*emia is the salient feature of T2DM, where * = glucose, TG's, cholesterol, NEFAs, uric acid etc. T2DM is a disease of chronic excess.
LCHF diet → ↓ Blood glucose & ↓ fasting TG's, but ↑ PP TG's, ↑ LDL-C, ↑ LDL-P & ↑ NEFAs. ↑ PP TG's & ↑ LDL-P are strongly associated with CHD risk.
Point 2: So?
Point 3 is wrong. A caloric deficit is essential, to reverse liver & pancreas ectopic fat accumulation. See Reversing type 2 diabetes, the lecture explaining T2D progression, and how to treat it.
Point 4 is misleading. Feinman doesn't distinguish between different types of carbohydrates. Resistant starch (amylose) is beneficial. See Point 11.
Point 5 is moot. Prof. Roy Taylor found that motivation determines adherence. Prof. Roy Taylor's PSMF was adhered to. See Point 3.
Point 6 is correct. Prof. Roy Taylor's PSMF is ~1g Protein/kg Bodyweight, some ω-6 & ω-3 EFAs & veggies for fibre. See Point 3.
Point 7 is misleading. Meta-studies e.g. Siri-Tarino et al and Chowdhury et al gave null results by including LF studies, also a dairy fat study which had a RR < 1 for increasing sat fat intake.
Point 8 is irrelevant. ↑ Dietary fat → ↑ 2-4 hour PP TG's. See Ultra-high-fat (~80%) diets: The good, the bad and the ugly.
Point 9 is partly correct. Microvascular, yes. Macrovascular, no. See Point 8.
Point 10 is mostly irrelevant. See Point 8.
Point 11 ignores results obtained with high-starch diets, where the starch is mostly amylose. See From Table to Able: Combating Disabling Diseases with Food.
Point 12 is misleading. The low-carbohydrate part is fine & dandy. It's the high-fat part that can be the problem. See Point 8.